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00:00:01
Hello everyone guys today we have our
00:00:04
last lesson recording everything else but
00:00:06
online and I really miss you
00:00:09
tomorrow we will meet as usual
00:00:12
well now let’s practice solving
00:00:15
problems the first task let’s look at it
00:00:17
and try to understand what
00:00:19
type of
00:00:21
person it is between the alleles of the gene absence of
00:00:24
sweat glands and hemophilia type A, crossing over
00:00:26
occurs,
00:00:29
hemophilia is linked to the x chromosome,
00:00:33
since with Dima, between the gene where the
00:00:36
cartoon is and the absence of sweat glands,
00:00:38
crossing over occurs,
00:00:40
this tells us that get the
00:00:42
absence of sweat glands is also located
00:00:45
on the x chromosome, and then it’s also beautiful it’s
00:00:47
true that it won’t happen, too, let’s not forget about him,
00:00:49
let’s not forget about the
00:00:53
disease not being specified, a
00:00:57
woman, a woman who does not have the specified
00:01:00
diseases,
00:01:02
whose father had hemophilia, whose mother had an
00:01:06
annual diploma, the absence of sweat
00:01:08
glands
00:01:10
means the mother was de homozygous
00:01:17
de homozygous and she had the absence of
00:01:20
sweat glands, which means she was and the
00:01:22
little one is small and homozygous, but
00:01:26
she didn’t have hemophilia, so she was already
00:01:28
big, the woman was
00:01:31
healthy in both respects, the father of this
00:01:35
woman, well, look, Matilda is
00:01:37
so much like that x why will this woman be a
00:01:40
little h big, the father of this woman
00:01:43
suffered from hemophilia then yes, he was x and
00:01:47
nothing much is said that he
00:01:50
had atrophy, lack of sweat glands,
00:01:53
so of
00:01:56
course, he gives his daughter exactly the
00:01:59
x chromosome, otherwise it’s not a daughter anymore,
00:02:02
but a big one, and
00:02:06
so it
00:02:09
turns out this woman
00:02:11
marries a man not having these
00:02:13
diseases x and the
00:02:16
big one is already big y let's see what they
00:02:20
will have what kind of gametes x the small one is
00:02:23
big x and the big one is even a little bit will
00:02:27
crossing over happen here
00:02:30
before crossing over will happen in
00:02:33
any case but will there be changes yes
00:02:36
if you change even the big one
00:02:38
small in some places, the gametes will
00:02:40
fundamentally change and the
00:02:44
children will become like this, the gametes that
00:02:46
were formed as a result of a beautiful lake,
00:02:48
but these two gametes were formed as a
00:02:51
result of the sci initial linkage of genes
00:02:55
born in this marriage, well, let's
00:02:57
find the shooting range of their offspring f1
00:03:01
we will get
00:03:04
forgiveness for more gametes already gametes the father was not
00:03:07
found because of the big big y now
00:03:11
let's find their offspring I should have
00:03:13
written here but I don't have enough space
00:03:16
so I write here a little higher and the
00:03:19
braid is small h big x big as much as big x as
00:03:23
small as big as big y
00:03:28
x and big as much as small x a
00:03:32
big-big and
00:03:33
x and big is even small y these children
00:03:38
were the result of linked
00:03:40
inheritance, the
00:03:41
remaining children
00:03:44
were the result of beautiful faith
00:03:46
will be numerically less than
00:03:59
a night, about numerically they don’t ask here, we will
00:04:02
definitely indicate for all children
00:04:07
the tasks where the coupling with gender occurs, we will
00:04:10
definitely indicate gender and
00:04:13
sign the phenotype
00:04:16
let’s write for ourselves a long time ago it’s already given
00:04:20
x and big is
00:04:23
without the
00:04:25
absence of sweat glands x small
00:04:29
is the absence of sweat glands xh large
00:04:32
without hemophilia x h small this is
00:04:36
hemophilia then here will be
00:04:39
without the absence of sweat glands and without
00:04:43
hemophilia
00:04:45
here will be the absence of sweat glands and
00:04:48
there will be no Dima fillet
00:04:51
here
00:04:53
without the absence of sweat glands and without
00:04:57
hemophilia without the absence of sweat glands
00:05:00
but with hemophilia
00:05:02
healthy in all respects
00:05:04
[music]
00:05:07
from the absence of sweat glands from
00:05:10
hemophilia unlucky does not sweat and the
00:05:13
blood does not clot
00:05:16
[music]
00:05:18
here without the absence of sweat glands and without
00:05:21
hemophilia, this is a healthy boy,
00:05:24
completely
00:05:28
draw up a scheme for solving the problem,
00:05:31
indicate the genotypes and phenotypes of the parents,
00:05:35
otherwise this one needs to write a healthy phenotype for the Russian Federation,
00:05:38
wait even further further,
00:05:40
continuation of those born in this marriage,
00:05:43
Monna is homozygous, I am a healthy daughter, still
00:05:45
here, monokuma, a second healthy daughter,
00:05:48
right here this is, for example, a healthy daughter, and for us
00:05:52
it is homozygous for one trait and
00:05:55
hydra zygote in another way, where else is it like this,
00:05:59
this
00:06:02
is heterozygotes to die homozygote, everything
00:06:06
fits these two,
00:06:09
she comes out healthy men, they have a
00:06:11
child, Dima helix, so that
00:06:14
they have a child, hemophilia she must
00:06:16
be a carrier of the hemophilia gene, so out of
00:06:19
these two daughters we will choose which one we
00:06:22
will choose the second because she has the
00:06:24
hemophilia gene, which means we take this girl and
00:06:35
marry her to a healthy man
00:06:55
will this girl have a
00:06:58
singular correction, yes it will happen but
00:07:01
gametes will not fundamentally change if
00:07:04
you change as much as a big one as a small one in some
00:07:07
places you will still have a big one
00:07:09
and a big one as much as a small one and a
00:07:13
big-big one
00:07:15
I'm a big one as much as a small one 2 identical two
00:07:18
times identical gametes we do not write
00:07:20
so here we do
00:07:22
not indicate any crossover additional comets we immediately
00:07:25
write the descendants we get x
00:07:29
and the big one is already small x and the
00:07:33
big one is even
00:07:38
big x and the big one is even small y
00:07:59
without hemophilia without
00:08:03
absence of hemophilia healthy in
00:08:06
all respects and
00:08:08
here, too, is it
00:08:11
possible in the first marriage to give birth to a child suffering from
00:08:14
this
00:08:17
disease, that
00:08:20
is, two in the first marriage we
00:08:23
have a boy who had the absence of
00:08:26
sweat glands and hemophilia, both at the same time,
00:08:31
why explain the answer here they want to
00:08:34
tell you they want you to tell them that
00:08:38
ghetto like 2 genes are linked and
00:08:41
crossing over occurs,
00:08:42
crossing over leads to the appearance of
00:08:45
such gametes and when this gamete
00:08:47
is combined with a game we get an
00:08:50
organism we get a boy with this
00:08:53
genotype like this further in
00:08:56
humans alleles of chicken genes blindness,
00:08:59
aka night acid, oh number and red-
00:09:04
green tanto nismo on one chromosome,
00:09:06
so we have color blindness, night
00:09:09
blindness,
00:09:10
they are on the same chromosome, we
00:09:13
know that color blindness is linked to the
00:09:15
x-chromosome, ara blindness, and just didn’t
00:09:17
know one chromosome of that blindness is also on the same
00:09:20
one chromosome and there will be blindness
00:09:23
without the specified diseases, a
00:09:26
woman
00:09:30
whose mother had night blindness and
00:09:33
color blindness, that is, her mother had both
00:09:37
night blindness and
00:09:40
color blindness,
00:09:42
was to write and
00:09:43
[music] let’s
00:09:46
write immediately before for
00:09:49
color blindness is
00:09:53
always a recessive trait and
00:09:58
night blindness should be a recessive
00:10:01
trait
00:10:07
because that this is painful, it is
00:10:10
not very common, recessive
00:10:12
traits are less common than
00:10:14
dominant, so we chose
00:10:16
recessive,
00:10:18
look, this woman’s mother
00:10:21
was like that, so she
00:10:23
only gets one type of gametes and she
00:10:26
can only give her daughter a
00:10:27
small d small and
00:10:30
which one her father was a father who had the
00:10:32
indicated diseases, that is, he was like
00:10:35
this,
00:10:36
she married a man who was colorblind, it is not
00:10:40
said that he had night
00:10:43
blindness, so we will assume that he does
00:10:45
not have it, neither he is colorblind,
00:10:48
the man has gametes and to the kaida itself small
00:10:53
x big d big xa big you are
00:10:56
small and y in the offspring we get x
00:11:00
and small where small x and big and
00:11:04
small
00:11:05
x and small you are small y
00:11:09
xa highest d big and x a big de
00:11:13
small not last xa big for
00:11:16
big y be sure to indicate gender gender
00:11:19
should always be obtained 50 to 50 50
00:11:22
percent girls 50 percent boys
00:11:25
and this girl will be
00:11:29
healthy due to night blindness but
00:11:34
with color blindness
00:11:36
this girl is healthy due to blindness
00:11:40
and
00:11:41
from I apologize he has night blindness
00:11:43
and
00:11:44
color blindness
00:11:47
this girl
00:11:49
it was a boy this girl is healthy and
00:11:53
healthy and a boy healthy healthy
00:11:58
born here was crossing over
00:12:03
born in this marriage daughter colorblind
00:12:06
married a man
00:12:08
theorist look theoretically here it would be possible to
00:12:11
carry out crossing over but
00:12:14
do we need it
00:12:20
here they don’t say anything about it
00:12:23
here too we don’t have this information
00:12:26
look further born in this marriage
00:12:28
definitely colorblind married a man who does
00:12:31
not have these diseases,
00:12:34
we have only one colorblind daughter so far, let’s
00:12:37
assume that it’s her
00:12:39
now,
00:12:41
you know how we’ll find out whether we’ll paint a
00:12:43
guyver here or not, we’ll see further if
00:12:46
they give birth to some kind of
00:12:48
child, which is impossible
00:12:50
get from this girl here means we will
00:12:53
need to get
00:12:54
more crossover gametes and then we will get
00:12:58
four more children here and we will
00:12:59
take another girl but let's
00:13:01
try with this one
00:13:03
x a small
00:13:07
small d small x a big you
00:13:11
little
00:13:16
mouse married a healthy man by
00:13:19
all indications,
00:13:27
a man is
00:13:28
big porridge y and
00:13:32
in the offspring we get x and big and
00:13:40
small you are small x and
00:13:44
big
00:13:46
like I have here in your why I had to do
00:13:49
things I’m here
00:13:57
x and big and small x and big
00:14:00
yes big
00:14:05
x small you are small y x big for
00:14:12
small y half cash register
00:14:19
Now let's see in this family a
00:14:21
child was born with night blindness and color blindness, now
00:14:30
we have a child with night blindness
00:14:33
and color blindness,
00:14:38
so draw up a scheme for solving the problem,
00:14:44
indicate the genotypes and phenotypes of the parents, indicate the
00:14:46
genotype, we will also indicate the phenotype,
00:14:49
healthy woman,
00:14:51
healthy man, according to the first sign,
00:14:54
suffering from color blindness,
00:14:57
woman here is a girl healthy butt the
00:15:01
first sign suffering from color blindness
00:15:04
man
00:15:07
[music]
00:15:10
healthy healthy
00:15:14
genotypes and phenotypes sex of possible
00:15:16
offspring here everything here we will also indicate
00:15:18
the phenotypes healthy boy color blind
00:15:22
healthy healthy healthy healthy all
00:15:26
this was indicated is it
00:15:28
possible in a first marriage to have a
00:15:30
child with night blindness and
00:15:33
no color blindness
00:15:41
here is such a moment, if there
00:15:44
was no beautiful lake, then we say that no, it’s
00:15:49
impossible, the second option, we solve the
00:15:53
second option, if there was a crossing over,
00:15:55
because we didn’t have any clues about the
00:15:58
presence of a crossing over,
00:16:01
here we do a
00:16:03
small d large and x and a
00:16:06
large d small and then we have there will be
00:16:09
four more descendants and a braid small d
00:16:13
big x a big and small xa
00:16:16
small d big y
00:16:21
xa big for little x big and
00:16:24
small and x a big
00:16:29
you small y
00:16:35
indicate gender
00:16:39
phenotypes this is healthy and healthy with
00:16:43
chicken slip boy with night blindness
00:16:46
and
00:16:47
healthy on the basis of color blindness
00:16:50
the boy is healthy on the basis of night
00:16:53
blindness but with color blindness
00:16:58
healthy with color blindness is
00:16:59
also not a girl and a boy is a healthy
00:17:02
organism, but in this case there is a
00:17:04
possibility that a child will appear who
00:17:07
suffers only from blindness but he
00:17:11
will not have color blindness only on the condition that
00:17:13
there was a Kassin of the year so this and you need to write down,
00:17:16
I recommend doing this task in the following
00:17:19
way: write the first
00:17:21
option without painting Hoover and write the
00:17:24
first option if there was no crossing over, the
00:17:26
second option if
00:17:29
there was crossing over and
00:17:30
then solve the second crossing scheme and
00:17:33
then in the answer indicate that before the
00:17:35
possible birth of a child in
00:17:39
case there was a color ambition
00:17:48
further in a person, they or the genes for
00:17:52
optic nerve atrophy and the
00:17:54
red-green water of the body are
00:17:56
on the same chromosome, but the same thing begins again, the
00:17:58
large x is atrophy of the
00:18:02
photos, I ask for the reason, on the contrary, it’s great
00:18:05
for this sign, and the small x is
00:18:08
atrophy
00:18:09
xb large, this is color blindness
00:18:16
xb small
00:18:20
color blindness a
00:18:21
x would be big big it is healthy according to
00:18:26
this sign a
00:18:30
woman a
00:18:34
woman whose mother had
00:18:37
optic atrophy and color blindness that is,
00:18:39
her mother had a small
00:18:44
small d small and a small d
00:18:47
small
00:18:59
accordingly this woman gives this
00:19:02
girl only this such gametes the
00:19:06
father did not have the indicated diseases, that
00:19:09
is, he was a big d big and scream
00:19:12
means the girls he gave big to big
00:19:15
she married a colorblind man a
00:19:18
big d small y let's write the gametes x a
00:19:23
small you are small x a big d
00:19:26
big x big and small
00:19:30
y
00:19:33
offspring x can it happen here
00:19:36
we paint coffee can it be written is this a task
00:19:39
again no so let’s do it this way let’s
00:19:42
formalize it directly this is the first crossing
00:19:47
without a beautiful faith or y so if
00:19:50
crossing over doesn’t happen
00:19:55
doesn’t
00:19:56
happen
00:20:01
we get x and small
00:20:06
yes small x big and small
00:20:10
x
00:20:12
small d small y
00:20:16
x a large yes I am large x large and
00:20:20
small x a large d large y we
00:20:29
all indicate the gender and
00:20:35
sign the phenotypes
00:20:39
healthy with color blindness
00:20:42
girl boy
00:20:45
with atrophy and with color blindness
00:20:52
completely healthy girl and
00:20:56
completely healthy boy
00:20:59
born in this marriage daughter color blind
00:21:02
here she married a man the
00:21:06
second crossing did
00:21:23
not have the specified diseases the man
00:21:26
was healthy
00:21:31
we indicate the gametes
00:21:37
we find the offspring
00:21:42
I swapped them here but it’s
00:21:45
not important you can’t change places like
00:21:48
this you could leave it
00:21:58
here for sure crossing over it will
00:22:01
happen but it won’t be visible here
00:22:04
what they write in this family, a
00:22:06
child was born with optic nerve atrophy and
00:22:09
before a nismo the
00:22:11
girl is
00:22:13
healthy healthy the
00:22:16
boy with atrophy and with color blindness
00:22:21
the girl is healthy and healthy and the boy is
00:22:26
healthy with color blindness
00:22:30
so indeed there is one such
00:22:33
child was born everything coincides make a
00:22:37
diagram for solving the problem
00:22:39
indicate their genotypes and phenotypes of the parents
00:22:42
phenotypes must be signed before signing healthy
00:22:45
healthy healthy colorblind
00:22:49
girl healthy in atrophy but with
00:22:53
colorblindness boy man more precisely healthy healthy
00:22:56
in children everyone indicated whether it is possible in the first
00:23:00
marriage to have a child with
00:23:02
optic nerve atrophy and no
00:23:04
colorblindness and here we will write
00:23:07
the second crossing not 2 crossings the
00:23:10
second option
00:23:11
here is the first we write like this the first crossing
00:23:16
if
00:23:18
crossing over occurs
00:23:25
if crossing over occurs and we indicate
00:23:29
the woman x and small yes small x
00:23:34
big d more in general what are we
00:23:36
writing here we are rewriting everything the same only
00:23:39
in addition we are writing two more types of gametes
00:23:42
this is how it was in the previous problem
00:23:44
we additionally write two types of gametes: a
00:23:47
small d large and x a large for
00:23:51
small they will be beautifully correct and a
00:23:53
whole series of descendants that are formed as
00:23:56
a result of crossing over and there you
00:23:58
will get a child with optic
00:24:00
nerve atrophy with the absence of color blindness, you
00:24:03
answer like this and write down that if
00:24:04
crossing over occurs, then it is
00:24:07
likely that there is a possibility of the birth of
00:24:10
such a child with trophies but without
00:24:12
color blindness, the
00:24:13
crossing scheme will be similar to how
00:24:16
we indicated here,
00:24:19
so
00:24:22
on in humans, the alleles of the genes for night
00:24:25
blindness and hemophilia include hemophilia
00:24:29
type A and are located on the same chromosome,
00:24:31
well here again begins sex large is
00:24:34
healthy for night blindness x
00:24:36
small is blindness
00:24:39
xh large is healthy xh small is
00:24:43
hemophilia monk homozygous and a woman
00:24:47
does not have the indicated diseases, that is, a
00:24:49
woman
00:24:51
she is healthy mono homozygous for one
00:24:57
trait she was
00:25:00
homozygous for another heterozygous
00:25:03
mother whose mother had night blindness
00:25:08
so the mother was starting to be blind
00:25:14
about her
00:25:17
hemophilia, nothing was said, we’ll do it like
00:25:20
this, and the
00:25:21
father did not have the indicated diseases,
00:25:24
but he was, and the big one is big, which means
00:25:29
the father gave it to her, and the big one, even the
00:25:31
small one, the mother suffered from night blindness,
00:25:35
she will give it to her exactly, and the small one, and what kind of one
00:25:38
she is will give the second one as much as a big second
00:25:41
gene as much as a big only a small one right there
00:25:43
can be as much as a small one here maybe a
00:25:45
very big one is needed for this woman
00:25:47
to turn out homozygous she is already
00:25:51
heterozygous by the first sign then
00:25:54
by the second she should be homozygous
00:25:57
now now we have one mass here but
00:25:59
if we put a small one here,
00:26:02
then she would be heterozygous, which does not
00:26:04
coincide with our conditions, her
00:26:07
mother married a man with
00:26:11
hemophilia,
00:26:12
that is, everything is fine here after rubbing her
00:26:14
legs mafia trouble,
00:26:17
we’ll find comets here and a small h
00:26:21
big-big-big
00:26:23
you’ll be here we paint the year, maybe
00:26:26
it will be, but in any case nothing
00:26:27
will change
00:26:31
descendants x and the small one is already big x and the
00:26:37
big one is already small here x is small
00:26:40
h big y here x and the big one is already big
00:26:44
x and the big one is even small and
00:26:48
like that,
00:26:57
who can say it’s great there thank you
00:27:00
here we will indicate the gender we will
00:27:03
sign the genotypes healthy
00:27:06
healthy with blindness but healthy
00:27:10
healthy healthy healthy healthy
00:27:14
here good industrial Dima chemists no super
00:27:18
blindness with chicken blindness is starting blindness you
00:27:21
can still live in peace those
00:27:24
born in this marriage a healthy daughter
00:27:27
married to a man who does not have these
00:27:29
diseases here there are
00:27:31
2 healthy daughters, you need to choose one in
00:27:35
this family, a child was born with night
00:27:38
blindness and hemophilia, the
00:27:40
girl needs to be a carrier of the gene for both
00:27:43
night blindness and hemophilia, this one has
00:27:47
only hemophilia, and this one has the gene for both
00:27:50
blindness and hemophilia, so
00:27:52
this girl is suitable we choose this girl by
00:27:55
you the first is you crossing
00:27:58
this is the second crossing
00:28:03
we write here x and the little one attributed
00:28:07
why the genotype of this girl
00:28:09
she marries a healthy man
00:28:16
gametes
00:28:45
and indicate the gender
00:28:50
girl boy girl boy
00:28:54
indicate phenotypes
00:28:57
healthy healthy with blindness but great
00:29:02
great I’m healthy and a boy
00:29:10
so the boy turns out
00:29:14
healthy with hemophilia, but we needed a
00:29:16
boy with night blindness, Kim and Philly, this is
00:29:19
where we can make a krasin grief
00:29:22
and the gametes will really change here
00:29:25
then there will be a small
00:29:31
small h small 1 crossover on I
00:29:34
gamete and a big one even big one
00:29:38
crossover on I gamete these crossover
00:29:41
gametes now let's use them we get
00:29:43
x a small h small x a
00:29:47
big-big
00:29:49
x small h small y
00:29:52
xa big as big x big it's
00:29:56
big
00:29:59
so and finally we got a child
00:30:03
suffering from blindness and hemophilia, let's show
00:30:07
them all gender,
00:30:14
sign the phenotypes everywhere and
00:30:20
voila, everything is ready, indicate the genotypes and
00:30:24
phenotypes of the parents for the offspring,
00:30:27
explain the birth of a child suffering from these two
00:30:29
diseases in a family of healthy
00:30:31
parents, the fact is that a
00:30:35
beautiful number occurred, this led to the
00:30:38
appearance of such a gamete
00:30:40
x small h small and as a
00:30:44
result when these are gametes combined with the
00:30:47
y-chromosome,
00:30:48
the result was a boy from Egypt with such a
00:30:51
genotype and with such a phenotype, so the
00:30:54
next task in ducks is the crest trait is
00:30:58
tasty and the perennial quality is a dream and and are
00:31:01
not linked, well, like everything is fine in a
00:31:05
homozygous dominant state, and the
00:31:07
big, big crest gene tasty causes
00:31:11
death of embryos in this crest gene is tasty
00:31:14
then big and small is something like whether there
00:31:17
is a crest or not, it is not yet
00:31:21
entirely clear which of them is dominant
00:31:23
which is recessive plumage b-large and
00:31:27
would be small that we have
00:31:31
crossbreeding grab them
00:31:35
with normal plumage ducks and crested
00:31:40
drakes
00:31:44
with normal
00:31:46
plumage, you know the date, such
00:31:49
drakes are vodka males, some of the offspring
00:31:53
turned out
00:31:55
without a tuft
00:31:57
and
00:31:58
with silky plumage of
00:32:02
silky pyramids, look at this
00:32:05
sign does not appear in the parents, but
00:32:09
appears in the offspring, since this
00:32:11
trait was suppressed here, then
00:32:14
the holidays will be red sin and that means there is a
00:32:16
small one without a crest, and a small one is
00:32:19
silky plumage, then and a big one
00:32:23
is the presence of a crest, but only in a
00:32:25
heterozygous state,
00:32:27
because in a homozygous state, a
00:32:30
crest may develop in them but does not
00:32:32
die, both are large, this is normal and
00:32:35
the plumage now
00:32:39
we can write them the genotypes are crested,
00:32:42
that’s for sure, but the big one is small, why
00:32:45
can’t we choose the big one, and the big one,
00:32:47
because they are not dead, normal, big ones,
00:32:51
since we have silky ones in our offspring, it
00:32:53
would be big, it would be
00:32:55
small, by the way, with negligence, the same thing is the
00:32:57
second reason why we are writing
00:32:59
here the small one this is because she
00:33:02
is born without crests and the little one is
00:33:05
small, that is, she was the carrier of the
00:33:07
gene without a crest tasty here is the same thing and
00:33:11
oh no we get the Punnett lattice
00:33:26
thalac
00:33:31
here we need the Punnett lattice we will write
00:33:36
it
00:33:44
more accurately we will draw it
00:34:19
ok but we guess everything that should
00:34:22
turn out the ratio day 33 k1 but there
00:34:26
will be death and you may already
00:34:29
remember in the last lesson we said
00:34:31
when such a genotype causes
00:34:34
death
00:34:36
what should be the ratio what but
00:34:40
usually it is typical there the number is so
00:34:42
specific
00:35:15
and now cross out all of these
00:35:19
because we are not
00:35:22
signing the dead phenotypes this species crested
00:35:26
normal crested normally
00:35:31
crested normal crested silky
00:35:38
without crest
00:35:41
normal
00:35:47
approach and normal
00:35:51
crested
00:35:53
silky
00:35:55
without crest
00:35:57
normal and without crest choco eats and so
00:36:09
now
00:36:11
by crossing the resulting first
00:36:14
generations of crested ducks with normal
00:36:18
plumage and
00:36:20
[music]
00:36:23
drakes with the same genotype it turned out
00:36:26
2 phenotypic groups so when
00:36:28
crossing in the first generation
00:36:31
crested normal
00:36:33
homozygous
00:36:35
paths we take 2 crossings brem crested crested
00:36:40
then only large small
00:36:43
normax normal plumage homozygotes
00:36:47
they are here directly task of writing
00:36:49
a duck and
00:36:51
we cross them and
00:36:54
series them with the same genotype of
00:36:58
gametes a large large
00:37:02
small large a big b big
00:37:05
small big in the offspring we get a
00:37:09
big a big big big a big small b big
00:37:13
-big a big small big
00:37:17
big I
00:37:21
small small big big let's
00:37:23
sign the phenotype for
00:37:27
him, this dead one, don't sign anything
00:37:30
better, the phenotypes for the one who
00:37:33
dies in the first place are empty waste of
00:37:35
time secondly, he died, we don’t even
00:37:37
take it into account in the splitting,
00:37:39
this will be crested normal crested
00:37:44
normal
00:37:46
without crest
00:37:49
normal and
00:37:52
it was said that it was said that
00:37:56
we got 2 Finns typical groups
00:37:58
we really get crested normal
00:37:59
and without crests normal
00:38:01
draw up a scheme for solving problems determine the
00:38:04
genotypes of the parent individuals
00:38:06
genotypes and here it would also be worth signing
00:38:09
the phenotypes it was worth the
00:38:12
genotype parental genotypes and
00:38:14
phenotypes of the resulting offspring here from the
00:38:17
first crossing we have a
00:38:18
Punnett grid which has both genotypes and
00:38:20
phenotypes again do not duplicate this in the
00:38:23
second squeak with the second crossing this is
00:38:25
all indicated determine and explain the
00:38:28
phenotypic split in the first and the
00:38:30
second crossing, you say, since a
00:38:33
big, big one causes death, then in the
00:38:35
first crossing, wasps without
00:38:37
such a genotype die with such with such and with
00:38:39
such about the author and a split of 6 is obtained,
00:38:43
three to two to one; in the second
00:38:47
crossing, the organism with
00:38:50
such a genotype dies and we get
00:38:52
a splitting of two to one, such a
00:38:55
problem is interesting
00:38:57
and the last problem is the repetition
00:39:01
in Drosophila of dominant genes
00:39:04
that control the gray color of the body and
00:39:07
the development of bristles, that is, they immediately
00:39:11
tell you that gray paint is a
00:39:14
dominant gray color and
00:39:18
bristles are
00:39:20
also a dominant feature,
00:39:23
then what kind of body is small? black
00:39:26
body
00:39:28
b there are no small bristles and
00:39:31
it is said that these two genes are located on the
00:39:35
same chromosome,
00:39:37
what does this tell us that these two
00:39:40
genes are inherited linked, the
00:39:43
recessive alleles of these genes that
00:39:46
cause the
00:39:48
black color of the body and the absence of bristles
00:39:51
are located on the other to the malaga chn and
00:39:53
chromosome
00:39:55
what genotype and phenotype of
00:39:58
the offspring and in what
00:40:00
percentage can be expected from a cross
00:40:03
where heterozygous sulfur females have
00:40:07
developed bristles sulfur
00:40:10
bristles
00:40:12
with a black male without bristles
00:40:18
black I
00:40:20
ask you to forgive them they should be
00:40:24
without bristles
00:40:26
without bristles, provided that the female has 50
00:40:31
percent of gametes were a crossover, well,
00:40:34
let's look here at four types of
00:40:36
gametes,
00:40:38
how linkage will be indicated here, you
00:40:42
need to carefully read
00:40:44
the first and second sentences and you
00:40:47
will understand everything, now I will indicate all possible
00:40:50
types of gametes and we will understand what they are
00:40:52
linked, re-read again in the morning
00:40:55
production dominant genes
00:40:58
controlling the gray color of the development of the
00:41:00
bristles are localized in one chromosome,
00:41:02
that is, initially it is the dominant genes
00:41:05
in one chromosome, that is, they
00:41:07
are linked; the
00:41:09
recessive alleles of these genes
00:41:11
causing the black color are
00:41:13
absent are located in another
00:41:16
chromosome,
00:41:18
here is another chromosome, and therefore we can
00:41:22
conclude that the linked ones are a
00:41:25
large big I'm small would be small
00:41:28
and these two
00:41:30
were formed as a result of Hoover's beauty
00:41:34
here and we will be fifty percent
00:41:37
here 1 meter and small would be small in
00:41:41
f1 we get first we will find the individuals
00:41:44
that were the result of
00:41:45
linked inheritance and big
00:41:48
small big would be small and this is
00:41:50
a small small boo small would be
00:41:53
a little now let's find those that
00:41:55
resulted from Krasin Goda Rao
00:41:57
big small boo small would be
00:41:59
small I would be small small would be
00:42:02
big would be a little let
00:42:04
's sign their phenotypes sulfur with bristles
00:42:10
black without bristles now with crossovers
00:42:14
children
00:42:15
gray without bristles and
00:42:19
black with
00:42:21
bristles it is said that 50 percent the gametes
00:42:25
were crossover, that is, in fact, it
00:42:27
turned out to be exactly equal, half will be
00:42:30
linked half crossover, this is
00:42:32
very rare, but it happens this way,
00:42:35
these will be fifty percent of
00:42:38
these, although they are not crossover,
00:42:40
although usually we have less cursor ones,
00:42:42
but what do they want? by
00:42:45
genotype and phenotype,
00:42:48
what percentage of their offspring
00:42:49
can be expected from
00:42:52
such a crossing,
00:42:54
we will get bristles like this
00:43:02
with bristles
00:43:06
with gray bristles with bristles, there will be a quarter of these
00:43:11
because these will also remain 50
00:43:13
percent for reading they will be 25
00:43:15
percent like this
00:43:19
black without bristles there are
00:43:22
also 25 percent of them, the remaining
00:43:27
crossover of
00:43:31
25 percent,
00:43:37
also 25 percent, be sure to indicate
00:43:41
that these two were formed as a
00:43:44
result of linkage,
00:43:50
linkage of genes, and big for big and
00:43:55
small would be small, but these
00:44:00
two genotypes and phenotypes were formed
00:44:04
as a result of a
00:44:07
beautiful lake,
00:44:13
this is the problem I hope Everything was
00:44:17
clear to you, if you have any questions,
00:44:18
write in the general group, I will
00:44:21
definitely be in touch to answer your
00:44:23
questions about the problem, that’s all for now
00:44:27
until tomorrow

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